Crohn´s disease is a disease caused by the abnormal production of immune cells, and is often caused by a leaky gut or a chronic infection.
In a new study, scientists at Stanford University have found a gene called TAC2 plays a role in causing the disease.
It was previously thought that TAC1 was involved in the disease, but a new gene analysis of a patient’s stool found that the TAC3 gene is also important in causing it.
The study, published in the journal Nature Genetics, was conducted by researchers at the Stanford School of Medicine and the National Institutes of Health.
They examined the stool of a group of patients with Crohn`s disease and compared the stool with the stool from people with no symptoms of the disease and healthy controls.
When the researchers examined the patient’s genetic material, they found that they shared TAC-2 genes that are linked to Crohn�s disease.
They found that these patients also had a gene that was associated with a more severe form of the illness.
The researchers then conducted a genetic analysis on the stool samples from the patients and found that all of the TAKs were associated with Crohns disease.
Crohn’s disease causes inflammation in the gut, but the most common form is a type of inflammatory bowel disease, which includes Crohns disease, ulcerative colitis, and ulcerated small bowel.
The two types of Crohn disease are usually treated with antibiotics.
But because the most commonly used form of antibiotic treatment is sulfonamides, many people with Crohs disease don’t get the proper treatment.
The Stanford researchers wanted to see if a different type of antibiotic, known as TACs, could help with Crohens disease as well.
TAC is a gene found in the small intestine.
It is present in nearly all animals and humans, including humans, but it has never been associated with any type of disease in humans.
The scientists looked at the TACK2 gene, which codes for a protein that binds to the TAT, a type that has no known function.
They also looked at a gene linked to TAC, which is a protein called TACA1.
These TAC genes were found to be associated with TAC syndrome, which was the second form of Crohni�s.
In Crohnís disease, the symptoms include bloating, diarrhea, abdominal pain, cramping, and pain in the lower part of the stomach.
The disease also causes ulcers and ulcers-like conditions in the digestive tract.
When TAC proteins are found in blood or tissues, they can cause inflammation, leading to infections.
TACA2 is not a normal protein, but instead an immune-signaling protein that can help fight infections.
When it was discovered that TACA proteins were associated to Crohn’s disease, scientists initially thought that this gene was related to Crohs inflammatory bowel disorder.
The TACA gene was found to have an important role in the development of Crohs diseases.
It can be found in people who are genetically predisposed to Crohmns disease, and its absence in people without a genetic predisposition may contribute to the disease itself.
But TACA-2 also has a role, and it was found that TACT-2 was linked to the other forms of CroHns disease as a result.
TACT2 is the main protein involved in TAC signaling, and this protein was associated to TACT1 in the Stanford study.
TCA1 is the protein responsible for producing TAC.
In this study, the researchers also found that this protein had a role also in Crohn especialy in patients with TAK1 and TAK2.
This study adds to a growing body of evidence that suggests that TCA is involved in Crohnia, but also that TAK3 plays a larger role in Crohtans disease.
The research also raises some interesting questions about how TAC regulates the gut flora and can contribute to inflammation in Crohs.